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Modified Growth Releasing Factor aminos 1-29, usually referred to as Modified GRF (1-29) or “ModGRF(1-29),” is a synthetic analog of the endogenous peptide signaling hormone Growth Hormone Releasing Hormone (GHRH). Endogenously produced GHRH has 44 amino acids in its chain structure. A truncated synthetic form of GHRH called sermorelin or GRF 1-29 has 29 amino acids; Modified GRF (1-29) is further changed in that it has four substituted aminos in its chain that serve the purposes of preventing degradation and oxidation in manufacture and transport as well as in vivo, while also increasing binding affinity to the GHRH receptors.Learn More
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GHRP-6, sometimes called growth-hormone releasing hexapeptide, is a synthetic partial analog of ghrelin, a peptide signaling hormone and growth-hormone secretagogue. GHRP is an acronym that stands for “growth-hormone releasing peptide.” GHRP-6 is a robust hexapeptide unlike Sermorelin, the fragile 44 amino acid analog of endogenous hypothalamic GHRH.Learn More
Triptorelin is a GnRH agonist (referred to as GnRHa, gonadotropin-releasing hormone agonist, a decapeptide)
All products available for purchase are for research purposes only. These laboratory peptides are not for use in food products or as any type of drug. Triptorelin peptides are not intended to treat, prevent, mitigate or cure any disease or medical condition.
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Selank or selanc is a synthetic heptapeptide analog of the endogenous tetrapeptide tuftsin, with the sequence Thr-Lys-Pro-Arg-Pro-Gly-Pro. Selank is sometimes called tuftsin analog 7. Tuftsin was discovered by Victor Najjar in 1969 (Siemion and Kluczyk, 1999), is produced primarily in the spleen, and is a fraction of Immunoglobulin Gamma (IgG). Selank was first synthesized in Russia as part of efforts to find a “superagonist” version of tuftsin, because tuftsin . The effects of selank can be divided into two broad categories: neurological and immunomodulatory, with the former coming from selank’s effects on BDNF (brain-derived neurotrophic factor) upregulation and enkephalin preservation, and the latter coming from the stimulatory effect of selank on phagocytes, neutrophils, and other immune cells. Selank has shown anxiolytic, neurotrophic, nootropic, antidepressant, bacteriocidal, antitumor, and __ effects in vitro and in vivo as well as promise for various chronic and acute disease states. While selank’s actions may be viewed as belonging to two categories, the emerging field of “neuroimmunology” provides new explanations for the effects of immunomodulators on memory, cognition, and behavior.